Chinese and US scientists have found a receptor cell that could be effective in the treatment and prevention of Alzheimer's disease.
When the receptor cell Trem2 is activated, it stimulates immune cells against the protein that causes Alzheimer's disease, and the immune system's ability to resist disease is increasing.
In a study conducted jointly by Fucien Medical University, University of Shi'am, University of Texas, University of California, Sanford Burnham Prebys Medical Reconnaissance Institute, Fujian Medical University reported that when a newly discovered receptor, Trem2, is activated, the microglial immune cells against Alzheimer's toxic amyloid beta protein , causing an increase in the ability of micrographs to resist Alzheimer's and symptoms.
Scientists, who liken Trem2 to a kind of triggering cell, say that in order to prevent Alzheimer's disease, which is caused by the death of nerve cells or the accumulation of toxic proteins in the human brain, it is said that the power to resist the microglial disease induced by the recipient's cell is increased and thus the toxic proteins are eliminated.
NEW TREATMENT STRATEGIES CAN BE DETERMINED
Without the use of Trem2, microglianine amyloid plaque cleansing is less successful, experts note, with this new study, that Trem2 has learned how it works and will support treatment strategies to strengthen the link between Trem2 and amyloid beta in the future.
The study was also published in the Neuron magazine.
The "amyloid beta" protein, one of the earliest manifestations of Alzheimer's disease, is seen as the main component of the disease in the brain of patients.
There is still no effective treatment for Alzheimer's. Therapeutic attempts do not respond because the visible indications are too late, especially when Alzheimer's disease is diagnosed within 10 years after the onset of the disease.
It is known that the nervous disorders that cause Alzheimer's disease affect 47 million people in the world.